Quick Summary
- Eczema is more common in children living in developed, urban, high-hygiene, higher socioeconomic settings, and less common in poorer, rural, lower-hygiene environments with animal exposure and unpasteurized milk.
- These patterns suggest eczema is not bacterial/viral or contagious and is not caused by poor hygiene; environmental and genetic factors interact in ways that are still not well understood.
- Genetics increase risk: a parentās childhood eczema, or a family history of asthma or hay fever, correlates with higher eczema likelihood.
- Symptoms vary by age: infants often develop facial/scalp rashes around 2ā3 months, children get rashes in skin creases (elbows/knees/neck/wrists/ankles), and adults have rarer but very dry, scaly, itchy rashes that can spread and infect.
- Management focuses on symptom control and preventing infection: rule out contact allergies, moisturize regularly (including petroleum jelly), and reduce scratching with trimmed nails or cotton gloves; eczema often resolves on its own.
Given the following information, which of these people do you think is more likely to contract Eczema, a skin disease affecting primarily children, featuring rashes of dry, itchy, and scaly skin?
Child A:
Chad lives in the United States in the swanky section of a bustling city (Park Slope, Brooklyn, New York). His parents are wealthy and upper class, and his mother was 35 years old when she gave birth to Chad. Chadās parents make sure that he is always clean and well-cared for.
Child B:
Rami is not named Chad, but he does live there, one of the poorer nations on Earth. His family is poor even by Chad (the country) standards and is, socioeconomically speaking, lower class. They cannot afford to live in a city, instead living on a small farm where they have two animals; a dog, and a goat. They drink the goatās milk. Ramiās mother was 15 when she gave birth to him. They cannot afford to keep him clean or give him adequate medical care.
Obviously, you would say Rami is more at risk for eczema. Rami is poor and doesnāt have access to Chadās standard of medical care, his parents canāt afford to keep him as clean as Chadās parents do, and heās obviously more at risk of disease and infection. On top of his poverty, he lives in an undeveloped nation, putting him more at risk. Ā Living around animals probably doesnāt help, and neither does drinking unpasteurized milk. Not to mention the fact that Ramiās mother had him as a teenager probably means that Ramiās even more at a disadvantage, having a mother who may not be as equipped to properly take care of him.
In fact, however, Rami is much, much less likely to develop eczema. Every single detail of Chadās situation is actually correlated with higher incidence of eczema: Living in a city, in the developed world, in a high social class, with an older than average mother and a high standard of hygiene is in fact linked to increased incidence of eczema. And, on the other hand, every detail of Ramiās case is linked with a lower incidence of eczema: living in an undeveloped country, in a lower social class, with lower standards of hygiene, around animals, drinking unpasteurized milk, and with a younger than average mother are all in fact linked with lower incidence of eczema. Chad may have a better life than Rami in innumerable ways, but heās more likely to have eczema than Rami.
This is all quite counter-intuitive and puzzling. Generally, better living conditions make you more at risk of eczema?
What it does mean, first of all, is that eczema is not a bacterial or viral disease that can spread in unhygienic conditions, because unhygienic conditions actually make it less likely. Secondly, and more importantly, it means that environmental factors play a large role in eczema, those factors being the stuff we enumerated: Better living conditions, better hygiene, and maybe exposure to animals and milk and the age of the mother. Why those particular factors are the ones triggering eczema is poorly understood. It is hypothesized that in our hygienic and clean societies, our children arenāt exposed to certain allergens, donāt develop a resistance, and are thus vulnerable later on to the sorts of allergic reactions that cause the rashes that constitute eczema. This may or may not be true, and should not cause you to forsake hygiene for your child (Chad may have more risk for eczema, but Rami has a higher risk for West Nile Virus).
Itās not just environmental factors, though. Genetics also seem to play a large role. Firstly, If a childās parent had eczema as a kid, the kid is much more likely to get eczema as well. But not just that. A family history of Asthma or Hay Fever also seems to correlate highly with risk of eczema.
All of these facts, taken together, give us an idea of what eczema is and what it isnāt. It is not a bacterial or viral disease, it is not contagious, and it is not the result of a lack of hygiene or poor living conditions (if anything, just the opposite). Rather, itās a condition caused by environmental and genetic factors interacting in ways we donāt quite understand to produce a certain set of symptoms.
Those symptoms manifest differently for different age groups. As we have mentioned, eczema primarily affects children. Between 10 and 20 percent of children get eczema, compared to 1-3% of adults. 90% of cases occur before the 5th birthday.
Infants (defined for our purposes as ages 0-2) often get eczema around 2-3 months old and break out in dry, scaly rashes on their scalp, forehead, or face, especially the cheeks. Because infants do not have the ability to verbally express how itchy they feel, a telltale sign of eczema may be the infant in question rubbing itself against bedding or carpeting, trying to scratch themselves, which you might find adorable for half a second before realizing your infant is doing that because theyāre in pain. They also might have trouble sleeping, which makes sense; youād have trouble sleeping too if you had an itchy rash and no fine motor skills. These rashes may bubble up and ooze or weep fluid or can become infected because of the aforementioned rubbing and scratching.
Children (from ages 2 and up until puberty) have different symptoms than infants. Their dry, itchy, and scaly rashes appear mostly in the creases of the elbows and knees, the creases between the legs and buttocks, neck, wrists, or ankles. In time, these rashes can become bumpy looking (like permanent goosebumps), lighten or darken, thicken in response to constant scratching and develop knots. They can even become itchier over time.
Adults, who, to reiterate, make up a tiny fraction of eczema cases, manifests as very dry, very scaly (scalier than childrenās eczema) rashes that appears in the creases of the elbows or knees, the nape of the neck, or just all over the body, and can be especially noticeable around the face, particularly near the eyes. It can itch non-stop, can thicken, and can become infected.
What do you do about eczema?
As we have mentioned, eczema is a condition that results from a certain set of environmental and genetic factors that interact in ways that are still poorly understood. That means that unlike other skin diseases, where we can try and get rid of the thing that causes the disease, whether it’s a bacteria or a virus or oil build-up or what have you, with eczema, we donāt have that option, because changing oneās genetic makeup is still considered unethical and changing their entire living circumstances is impractical. So when treating eczema, we donāt so much address the causes of the symptoms than the symptoms themselves.
Thereās one exception to this, which is allergic reactions. Skin reactions to allergens and eczema are very similar. You might even say that eczema is best defined as āthe skinās allergic reaction to something we donāt know aboutā. So the first response to a case of eczema, particularly if it’s in an adult, is to try and figure out whether it’s eczema or just an allergic reaction. People have allergic reactions to all sorts of things; certain laundry detergents, gold jewelry, fake jewelry, certain fragrances. If the rash only appears in a certain area touched by something that might be an allergen, try removing the allergen from the equation. That would be the easiest and most straightforward way of dealing with the problem.
If that doesnāt work, weāre back on symptom management. That means, primarily, two goals:
- Get rid of the rash
- Prevent infection
To get rid of the rash, and reduce dryness and itchiness, use moisturizer. Which moisturizer? Pick one. Just go down the aisle at your local pharmacy and pick one. Different brands work best for different people, and some trial and error may be necessary for determining which brand is best for you or your child. For a cheaper and fragrance-free option, consider using petroleum jelly. Apply the moisturizer at least twice a day, or when you or your childās skin seems dry.
To prevent infection, really try your best to keep from scratching. When you scratch, you create holes in your skin that bacteria can get inside of, particularly when your skin is damaged as it is. This is a hard job for adults, to say nothing of children, so with children, some tricks may be necessary. Make sure their fingernails are short and smooth, to limit scratch damage. At night, putting cotton gloves on your child may prevent them from scratching during sleep.
The good thing is, eczema usually goes away on its own, even if it can be frustrating and painful while itās present. If itās any comfort, bear in mind that if you or your child has eczema, because of the environmental factors we mentioned at the beginning of this article, your life is in all likelihood pretty good in all the other areas besides ānot having a dry, itchy, scaly rashā.